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Individual patient differences such as age, metabolic status such as diabetes, obesity, chronic inflammation levels, and medication history can significantly impact the GPX4/GSH antioxidant axis and iron homeostasis balance

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From both the docked complexes, it is observed that the glutathione parts of the adducts make H-bond and/or electrostatic interactions with the key glutathione binding site residues S40, R44, R72, R114, E115, and S172

Traves, S

Unlike other compartments, the ER lacks the reductase needed to convert GSSG back into GSH, making GSSG export vital for maintaining the GSH/GSSG balance