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Non-mitochondrial ROS sources in diabetes Regardless of whether excessive ROS formation is the primary initiator or accompanying factor in the pathogenesis of diabetes mellitus, oxidative stress plays a key role in the organ complications such as cardiovascular problems, neurological problems (stroke), diabetic retinopathy, nephropathy, and neuropathy

7 Epigenetic regulation strategies Chronic stress and inflammation can lead to epigenetic alterations in genes related to synaptic plasticity and inflammation

Lactate could increase the concentration of the components of the malateaspartate shuttle, which increases the rate of transport of reducing equivalents to the mitochondrial matrix [178]

Particularly noteworthy, mechanical overload can activate the mechanosensitive ion channel Piezo1, mediating calcium influx, which subsequently triggers the downstream GPX4-regulated ferroptosis pathway (Yao et al., 2021