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Targeting -Synuclein aggregation and its role in mitochondrial dysfunction in Parkinson's Disease

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10.1186/s12933-025-02751-3 135 SpositoA

Mice that overexpress Jak2VF in their erythroid lineage (VFEpoR mice) exhibit heightened necrosis in atherosclerotic plaque and erythrocytic ferroptosis, and VFEpoR increases erythrocyte lipid hydroperoxide and decreases antioxidant defense ability