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These active components help reduce abnormal -syn aggregation, block its neurotoxic effects, inhibit the apoptosis of dopaminergic neurons in the substantia nigra, regulate microglial activation, reduce the release of pro-inflammatory factors such as TNF- and IL-1, enhance antioxidant defense systems (e.g., by upregulating the Nrf2/HO-1 pathway) to mitigate oxidative stress damage, and promote neuroregeneration and synaptic repair

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10.1186/1471-2202-9-27 BMC Neurosci

Haemostatic effects of levothyroxine and selenomethionine in euthyroid patients with Hashimotos thyroiditis