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Notably, one promising avenue involves leveraging the astrocytic urea cycle to enhance neuroprotection and facilitate a liver-like clearance mechanism in the brain, offering potential strategies for mitigating neurodegenerative processes 59,73

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A previous study showed that the binding of antiestrogens to ER and particularly to ER can induce the expression of the antioxidant NQO1 74

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P=0.046), and higher hazards of transplant-free mortality (adjusted HR 2.3, 95% CI 1.23, 4.38