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Consistent with reports showing that the topoisomerase II-inhibitor Etoposide is a substrate of ABCC1 42 , the GI 50 of MOLM-13 ABCC1 -knockout clones to Etoposide was 1.4 2.4-fold lower than in control cells (Supplementary Fig

This, in turn, affects enolase 1 (ENO1)-mediated glycolysis, ultimately contributing to the development of myocardial fibrosis (58)

Wei Y, Lu C, Chen J, et al

10.1016/S2468-1253(20)30122-9 117

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