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Hyperthyroidism and Thyrotoxicosis

10.1016/j.biopha.2024.116444 Biomed

It promotes TH17 cell differentiation

These findings underscore the potential for developing additional therapeutic targets beyond MAOB inhibition

Specifically, it increases the pro-apoptotic protein (Bax, Bim, Bad and Bak) expression, while decreasing anti-apoptotic protein (Bcl-2 and Bcl-xL) expression, and activates Poly (ADP-ribose) polymerase 1 (PARP1) and Caspase-3, ultimately triggering cell death [143]