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However, in Be(2)-C cells, it reduced ATP content by more than half ( Figure 3D ) without increasing cell death ( Figure 4A ), indicating a cytostatic rather than cytotoxic effect

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These medications may reduce how long B12 remains in the body, requiring individuals to supplement more frequently to maintain appropriate levels

SREBP1cPAX4 axis mediates pancreatic beta-cell compensatory responses upon metabolic stress

Vitamin B12 plays a critical role in red blood cell production, nervous system health, and metabolic function