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Pts were divided into two groups according to the cytogenetic findings: high risk (monosomy 7 and/or complex karyotype) in 38 children (64%) and low risk (others cytogenetic abnormalities and normal karyotype) - 21 (36%)

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Finally, we showed that pharmacological inhibition of the thioredoxin system by the chemotherapeutic drug arsenic trioxide (ATO) enhanced MTX cytotoxicity in vivo against one out of four primary human ALL cells, suggesting a novel therapeutic rationale to be further explored against ALL