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Patients with MDS may have aberrant expression of antigens on their erythroid precursors, such as CD71, which may be useful in distinguishing MDS from other causes of persistent anemia

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Furthermore, ammonia has been observed to activate SREBP-1, thus establishing a feed-forward loop that promotes glutamine metabolism and lipid synthesis, as well as tumor cell proliferation [24]

Introducing external piR-FTH1 reduced Fth1 mRNA via HIWI2 and HILI, increasing TNBC cells doxorubicin sensitivity by 20-fold

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