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In these situations, vitamin B12 is the priority treatment, as untreated deficiency can lead to irreversible neurological complications

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Under zinc-deficient conditions, mutant SOD1 can generate highly active hydroxyl radicals and peroxynitrite via abnormal copper chemistry, further exacerbating neuronal oxidative damage and protein nitration (120)

To combine data across different replicates, we performed fixed-effect and random-effect meta-analyses for each parameter in the best model ( Supplemental Table S13)

it may prevent the absorption of lead as well as inhibit its cellular uptake and decrease its cellular toxicity