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Aging is not a single-pathway problem

J Ind Microbiol Biotechnol

6e and Supplementary Fig

It also pairs well with GHK-Cu in patients focused on skin, tissue repair, and systemic cellular regeneration alongside the longevity mechanisms Epithalon provides

The proposed mechanism centers on modulation of the nitric oxide system: BPC-157 counteracts L-NAME-induced eNOS blockade in rat models and supports vascular sprouting through VEGFR2 internalization (Hsieh et al., J Mol Med , 2017)