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While it promotes healthy collagen, it also downregulates pro-fibrotic factors and excessive collagen synthesis

Tumor redox heterogeneity, variations in intracellular GSH levels, and discrepancies between in vitro and in vivo redox environments make precise prediction of therapeutic activation difficult

Gene and protein characterisation of the human glutathione S-transferase kappa and evidence for a peroxisomal localisation

The absence of smad4 was also found to enhance mitochondrial ATP synthase inhibitory factor (ATPIF1) activity, ATP synthase inhibition, and reduce mitochondrial membrane potential, ultimately promoting glycolysis by decreasing glucose metabolic flux and increasing glucose accumulation in the podocyte [62]

Steffen M, Sarkela TM, Gybina AA, Steele TW, Trasseth NJ, Kuehl D, et al