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Nevertheless, they are limited to detecting known resistance gene targets, cannot confirm the actual phenotypic expression of resistance, and are even less capable of identifying unknown or newly emerging resistance mechanisms [14, 15]

For non-hematological AEs using multivariate logistic regression analysis, the C/T genotype group of MTHFR_a , high serum RBP level before pemetrexed treatment, high serum B12 level before premedication, and high red blood cell (RBC) count before pemetrexed treatment (Supplementary Table S3(a)) were the crude low-risk factors for G2G4 non-hematological AEs in the first cycle

Proper use of modifiers ensures that claims for B12 injections are processed accurately, without unnecessary denials or delays

Fatty acid synthase inhibitor TVB-2640 reduces hepatic de novo lipogenesis in males with metabolic abnormalities

Chapter Fifteen Vitamin B12 deficiency