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H., Pretorius, I

164 This disorder is an autosomal recessive condition and, in the family studied, presumed carriers had reduced GCS activity, although erythrocyte GSH levels were normal

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The requirement of two distinct glutathione interaction sites for the efficient reduction of GSSR by GSH explains the deviating properties and substrate preferences of glutaredoxin subfamilies as well as thioredoxins with implications for the design and optimization of artificial enzymes and inhibitors