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10.1016/0169-328X(93)90072-W Mol

Recent investigations have unearthed a novel player in the ferroptosis domain, MBOAT2, functioning autonomously of GPX4 and FSP1, overseeing ferroptosis surveillance through phospholipid remodeling and proficiently impeding ferroptotic processes

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However, the H 2 O 2 -induced increases in PTGS2 , SLC7A11 , and TNF mRNA, surface expression of the transferrin receptor, and lipid peroxidation are features that accompany ferroptosisan iron-dependent form of cell death

This reaction is reversible and can adapt to higher levels of homocysteine, converting it back to SAH