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RJM would like to give special thanks to all the people who helped fund this work

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These metabolic and proteomic changes collectively indicate that in ATP6AP1 KO cells show a metabolic phenotype protected from ACT-induced rerouting of carbons, suggestive that partial v-ATPase disruption appears to reduce cellular reliance on mitochondrial oxidation, enabling resistance to mitochondrial insults while limiting biosynthetic capacity

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