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5.2.2 Limitations due to the disease The pathophysiology of mutations or mechanisms underlying the neuronal pathology of mitochondrial diseases is still unknown in most patients, which makes it difficult to interpret the mitochondrial or neural readout in pharmacological screenings

Effects of dietary components on testosterone metabolism via UDP-glucuronosyltransferase

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near the start, the protease may strip it Glutamine at any position contributes more than its share because P1 Q is the most stringent feature recognized Crystal structure: The TEV S1 pocket is deep and Gln-specific (Phan et al., 2002), but the S2S6 pockets are shallow and water-mediated, explaining the tolerance (Phan et al., 2002)

In cotton, GhSPDS11 has been characterised as a key spermidine synthase isoform induced by alkaline stress