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We point out the potential parallels between ALD/AMN and inflammatory relapsingremitting/primary progressive MS, also at times manifesting as highly aggressive tumefactive disease (Marburg variant)

Downregulation of fatty acid oxidation led by hilpda increases G2/M arrest/delay-induced kidney fibrosis

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In summary, age-dependent changes such as increased catalytic iron levels, weakened antioxidant defenses, elevated mitochondrial-derived oxidative stress and sustained membrane vulnerability to peroxidation, collectively place neurons in the aging brain in a fragile state

[4] The coordination chemistry is what makes this possible