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The lethal accumulation of lipid peroxides is a major feature of ferroptosis and involves the confrontation between intracellular ferroptosis execution and the ferroptosis defense system

However, there are some limitations in these modalities, such as a lack of appropriate disease models and systemic interactions, such as neurohumoral regulation and immune cell involvement, restricting their translational potential [65]

"Association between Serum Vitamin A and E Levels and HIV-1 Disease Progression." AIDS 11.5 (1997): 613-20

(1989) Reduced glutathione effects on -tocopherol concentration of rat liver microsomes undergoing NADPH-dependent lipid peroxidation

These ROS are intricately linked to cell damage, inflammation, carcinogenesis and aging (32)