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investigated Thymosin Beta-4s role in accelerating wound healing in preclinical models, noting actin-mediated cell migration as a key mechanism

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We note that inherent biological parasite differences must be considered to accurately model malaria dynamics in co-endemic regions and recommend our model for use during sub-national tailoring of interventions

Results NAFLD development was associated with TNF overproduction and perturbation of glutathione homeostasis in the liver Long-term HFD consumption in mice represents a well-established animal model of NAFLD

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