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MxB was expressed in IFN-2-treated cells transfected with control siRNA, where it accumulated at the nuclear envelope as well as in the cytoplasm, as previously described 10,47

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Alvarez de Sotomayor M, et al

Due to its functional profile, oxytocin was proposed as a potential contributor to the ASD pathophysiology as early as the late 20th century, [35, 36], prompting interest in therapies targeting oxytocinergic signaling for the core ASD symptoms

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