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The authors concluded that GH-excess is able to promote severe glomerular damage in mice in the absence of IGF-1, whereas tubular hypertrophy observed in GH-transgenic mice is most likely mediated by concomitant IGF-1 excess

However, the differences in methodologies and experimental design prevent direct comparison between these studies

Melatonin administration to aged mice also demonstrated the therapeutic utility of this compound in the treatment of age-related cardiac sarcopenia, since it preserved the normal cardiomyocyte structure and restored their number, as well as to reducing -myosin heavy chain expression (a marker of cardiac hypertrophy) in these animals

In some architectures, the IdP and RP do not communicate directly with each other

Science 278, 5258