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By midlife, NAD+ can drop by 50%, exacerbated by inflammation and lifestyle.[14] Factors like poor diet, sedentary behavior, or chronic stress further deplete NAD+ by activating consuming enzymes, creating a vicious cycle

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The development of tracers that target phenotype-specific markers, such as those associated with trophic factor release, phagocytic function, or anti-inflammatory signaling, would represent a major step forward in evaluating therapeutic efficacy and disease progression