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Known as the bodys "master antioxidant," it plays a central role in protecting cells and supporting detoxification

Cell fraction was collected and processed as described above

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While low dose endotoxin triggered NAMPT-dependent NAD + salvage pathway to support pro-inflammatory response, high dose endotoxin drove a switch from NAD + salvage to IDO1-dependent NAD + de novo biosynthesis at late stage, leading to persistent anti-inflammatory properties and immune tolerance [98]

As cysteine stabilises CDO levels, cells that express high levels of NRF2 are sensitive to CDO-related toxicity