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Neohesperidin protects against colitis-associated colorectal cancer in mice via suppression of the NF-B/p65 and MAPK pathways

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Under normal oxygen conditions (normoxia), HIFs are usually hydroxylated by prolyl hydroxylase (PHD) domain enzymes, which modify HIFs for ubiquitination and subsequent degradation by the proteasome, ensuring that their levels remain low [1]