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These proteins detect DNA damage in the nucleus and play key roles in maintaining genomic stability

These nucleic acid molecules can function as full-length nucleic acids, as further described herein

however, in specific genetic contexts, its abnormal splicing or functional inactivation may lead to insufficient intracellular iron accumulation, thereby limiting Fenton reaction efficiency and attenuating radiotherapy-induced lipid peroxidative damage (196).Conversely, overexpression of ACSL3, a member of the long-chain fatty acid-CoA ligase family, enhances the activation of polyunsaturated fatty acids (PUFAs) such as arachidonic acid (AA), promoting the synthesis of oxidation-sensitive phospholipids like AA-PE and significantly increasing membrane susceptibility to radiotherapy-induced lipid peroxidation (197)

Radiation sensitization of HER-2/neu overexpressing head and neck cancer using EIA gene therapy

- i vi da nhy cm: Nn kim tra kh nng kch ng ca sn phm bng cch th dng kem dng trn vng da nh nh sau tai, di cm trc khi p dng cho ton mt