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IGF-2R has a strong affinity for IGF-2 but shows minimal binding to IGF-1 and does not interact with insulin [66]

While preclinical and translational studies indicate that disrupting mucin disulfide bonds can relieve mucus plugging and AHR in animal models and ex vivo human tissue [70, 71], no RCTs have demonstrated the clinical efficacy of mucolytics in asthma patients