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Description

With the help of cell cycle arrest, DNA repair pathways are subsequently activated to inhibit cell cycle progression, providing a window of repair for the cell

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These include use of immunomodulatory agents as well as inhibitors of B-cell receptor signaling that target either Bruton tyrosine kinase (ibrutininb) or PI3 kinase delta (idelalisib).50 Pro-oxidant based therapies utilize oxidative stress as a cellular weakness of leukemic cells and represent an example of a synthetic lethality approach

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A relation between gastroduodenal muscle contractions and gastric emptying

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