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The MDA and mitochondrial LPs increase in a DOX concentration-dependent manner

We discuss both approaches below

335 The upregulation of DLAT is associated with mitochondrial dysfunction, activation of the PI3K/mTOR pathway, autophagy suppression, and eventual cell death

Indeed, many studies highlighted the potential therapeutic effects of p38 MAPK inhibitors in both asthma and COPD (Kim et al

However, mitochondria are the powerhouse of the cell, playing a crucial role in energy production, regulation of metabolic pathways and initiation of apoptosis