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Current hypotheses for the spread of cell damage in neurodegenerative conditions, such as a prion-like spread of aggregated protein, are not able to explain these extra-CNS phenomena, whereas toxic metals can account for both CNS and associated non-CNS disorders

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In some instances it seems likely that GSTs are induced by the general oxidative stress caused by these diverse treatments, but in other cases GST induction is specific to the particular stress [27]