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Its pathogenesis involves a complex interplay of immune dysregulation, primarily a T-helper 2 (Th2) and T-helper 22 (Th22) skewed response, and epidermal barrier defects ( While clinical evidence is currently lacking, the pharmacological profile of T

Consequently, an error in either nDNA or mtDNA encoding for proteins that make up any of the subunits could result in disorder(s) of the ETC and the mitochondria

doi: 10.1002/j.1460-2075.1995.tb00038.x

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Fibroblasts produce matrix metalloproteinases (MMPs) that degrade ECM proteins 51