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Oxidant stress, mitochondria, and cell death mechanisms in drug-induced liver injury: lessons learned from acetaminophen hepatotoxicity

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In view of finding that BPH had an inflammatory component and that therapy with cetrorelix caused an improvement in LUTS in men with symptomatic BPH, our group investigated the mechanism of action and effect of cetrorelix in a male Wistar rat model of BPH 45

Heme oxygenase-1-modified BMMSCs activate AMPK-nrf2-FTH1 to reduce severe steatotic liver ischemia-reperfusion injury

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