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Results Proteomic analysis of chronically cigarette smoke extract (CSE)-exposed phBECs confirms known CS-regulated pathways and suggests NUPR1 as a potential novel regulator of the airway epithelial response to CS Label-free LCMS/MS-based proteomic analysis of control phBECs of the pooled data of four time points of differentiation (day 7, 14, 21 and 28, see Fig

Practice guidelines on the diagnosis, surveillance, and therapy of Barretts esophagus

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Gene expression was visualised through green fluorescence from enhanced green fluorescent protein, while the blue fluorescence of PCDs confirmed their utility as intrinsic makers for gene delivery and bioimaging [96]

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