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LPS-induced ALF mice exhibit neutrophil infiltration and significant formation of neutrophil extracellular traps (NETs), exacerbating liver oxidative stress and inflammatory damage[44-46]

Mechanistically, it improved mitochondrial function by reducing respiratory inhibition and mitochondrial calcium overload, which correlated with increased ATP levels in tissues

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doi: 10.1038/s41575-020-00385-2 70

52,53 In DFU, the increasing expression of IL-6 results in a sharp increase in phosphorylated STAT3 levels, 54 which mediates impaired immune cell activation, recruitment, and survival, resulting in delayed wound healing