glutathione gsh biosynthesis pathway Laboratory evolution of reveals natural compensatory pathways Scheme representing the biosynthesis and

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PX-12 achieves a reduction in TXN1 activity either by covalently binding to the key cysteine residue Cys73 in TXN1 or by enhancing the dimerization of its oxidized form [225]

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However, this alteration in metabolism makes these MYC-driven tumors hyper-sensitive to oxidative stress [1]