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These verdicts characterize the regulation of sortilin shedding and identify a novel mechanism by which sortilin ectodomain shedding acts as a regulatory switch for delivery of BDNF to the secretory pathway or to the lysosome, thus modulating the bioavailability of endogenous BDNF (Fig

confirmed no cytotoxicity to HaCaT keratinocytes or human dermal fibroblasts across a six-log concentration range (0.0058 to 5800 micromolar) 3

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Without long-term human studies, its safety profile remains largely unknown

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