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Sharabi et al

Transformation of any part of the gastrointestinal (GI) tract can have adverse implications on nutrient absorption, with the loss of parietal cell mass that occurs post-gastrectomy responsible for VB12 deficiency

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Vitamin B12 in foods is released, which leads to a significant decrease in protein-bound vitamin B12.[1] Figure 1: Processing and absorption of vitamin B12 Vitamin B12 starts by binding R protein or haptocorrin in the mouth, which travels to the stomach to get hydrolyzed by HCl from parietal cells