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p53, as an effective oncogene, promotes ferroptosis mainly through the canonical and non-canonical pathways (Dixon et al., 2012)

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However, challenges remain with AHR agonists, particularly because increased CYP enzyme activity induced by AHR activation can affect their absorption and entry into the circulatory system [10]

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Roth, 2008

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BarendsenRWDijkstraIMEVisserWFAldersMBliekJBoelenAet alAdrenoleukodystrophy newborn screening in the Netherlands (scan study): the X-factor

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6.3 Assessment of total antioxidant capacity (TAC) CF pathophysiology involves disrupted cellular equilibrium alongside persistent inflammatory responses that drive progressive tissue deterioration, with particular severity in pulmonary manifestations (OSullivan and Freedman, 2009)

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