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Moreover, pharmacokinetic constraints-including limited bioavailability and uncertain brain penetration-must be considered when interpreting gene-expression effects observed in vitro

Differential methylation in ID-associated genes (e.g., COLEC11 , SHANK2 , GLI2 , and KCNQ2 ) and imprinted genes (e.g., FAM50B and MEG3 ) suggests that some ID patients may have undiagnosed imprinting disorders (Kolarova et al., 2015)

It boosts autophagic and apoptotic cell death by altering various AMPK and Akt signalling and subsequently increasing the levels of autophagic markers [225, 228]