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found that disrupting cellular glycolysis can promote intracellular Cu overload, convert the TME from a suppressive state to an active state, and enhance the ICD induced by cuproptosis, thereby inhibiting tumor growth and distant metastasis, which was achieved using a nano-platform in animal models (Li Q

Martinot-Peignoux, M

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[6] After deep intramuscular injection the lipophilic ester diffuses slowly from the oil depot, undergoes enzymatic cleavage to free testosterone and enters the systemic circulation, where roughly 98 % is reversibly bound to sex-hormone-binding globulin and albumin while the unbound fraction translocates across cell membranes to activate androgen receptors