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Recently, overexpression of ATF4 was reported to be prominent in a wide variety of tumors and to protect tumor cells against multiple stresses, as well as a range of cancer therapeutic agents [11], [12], [13], [14], [15], [16], [17]

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In ALS skeletal muscles, the increase in the activity of malate dehydrogenase and cytochrome C is accompanied by a decrease in citrate synthase activity, activating a compensatory mechanism to maintain the production of oxalacetate in the muscles, and thus the Krebs cycle

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NF-kappaB inhibitors for the treatment of inammatory diseases and cancer