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Furthermore, the established role of ROS in modulating tyrosine phosphorylation during capacitation and other relevant processes is tightly regulated by complex cross-talks between various signaling pathways, including the cAMP/PKA and the extracellular signal-regulated kinase pathways (7981)
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N-terminal acetylation of -synuclein slows down its aggregation process and alters the morphology of the resulting aggregates
