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Pretreatment with a tyrosine kinase inhibitor or with a serine/threonine kinase inhibitor inhibited both the oxidative burst and the induction of GST in the incompatible interaction (Rajasekhar et al., 1999)

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Recombinant Rha1 and Rha2 metabolize 4-HNE and ectopic expression confers 4-HNE resistance to sensitive bacteria In order to determine if these putative enone reductases can directly utilize 4-HNE as a substrate, we generated recombinant Rha1 and Rha2 proteins

1), upon acute stimulation we also observed a decrease in PDHC and OGDC activity and their functional lipoic arms starting soon after iNOS induction and peaking at 24 h post-stimulation