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798 This mechanism restores mitochondrial targeting sequence pre-processing and alleviates feedback inhibition of mitochondrial processing peptidase activity in fibroblasts from PITRM1-deficient patients, improving Frataxin maturation and mitochondrial function

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We have, therefore found a critical role for the branched ubiquitin chains in shifting the equilibrium between ubiquitin conjugation and deconjugation

This oxLDL is then engulfed by macrophages derived from the migrating monocytes, ultimately forming the characteristic foam cells associated with atherosclerosis (72, 73)