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The pivotal role of FDX1 in this pathway is underscored by experimental evidence that genetic ablation of FDX1 confers significant protection against DOX-induced cardiotoxicity in mice, preserving the left ventricular ejection fraction [76]

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These monotherapy results established proof of concept that amylin receptor agonism alone can produce clinically meaningful weight reduction and informed the dose selection of 2.4 mg for the combination product CagriSema

IFN- triggers an intracellular signalling pathway in the mitochondria called the itaconate pathway

So yeah, you could bump that up certainly with an injury because probably you do need proportionally more again, depending on the degree