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In ALS, pathological intronic hexanucleotide GGGGCC expansions in the C9orf72 gene are thought to contribute to neurodegeneration by multiple mechanisms, including toxic cytoplasmic dipeptide repeat protein aggregates, toxic RNA gain-of-function, or loss of function due to haploinsufficiency [20, 21]

The effect of hexamethonium was not different in AdrX WKY, whereas hexamethonium increased overflow in AdrX SHR ( P 0.011 compared to the PBS- or AdrX + PBS-pre-treated SHR groups)

These forms of vitamin K are designated menaquinone-n (MK-n), where n stands for the number of 5-carbon units (MK-2 to MK-13) (2, 3)

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