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These results suggested that AG has the potential to treat DCM by reducing inflammation and oxidative stress, and its mechanism may be related to NF-B and NOXs/Nrf2 signaling pathways

GPx-1 prevents the accumulation of hydrogen peroxide and thereby protects against oxidative stress

The antiinflammatory mechanism of methotrexate depends on extracellular conversion of adenine nucleotides to adenosine by ecto-5-nucleotidase: findings in a study of ecto-5-nucleotidase gene-deficient mice

Heterozygous knockout of p85 improved insulin sensitivity in mice and complete absence of the p85 isoform resulted in hypoglycaemia

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