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However, only REST deficiency, rather than Fer-1, restored the downregulated GCLM expression and reduced GCL activity in AKI mice (Figure 7, C and D), indicating a unique role of REST and the different mechanisms of ameliorating ferroptosis between REST deficiency and Fer-1

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Wohlford JG, Wright JC

This effect is achieved by the increased ROS formation (prooxidant effect), resulting in caspase 8 cleavage, inhibition of Janus kinase 2 (JAK2), and triggering regulator of apoptosis BAX, known as bcl-2-like protein 4

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