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Sergi D, Boulestin H, Campbell FM, Williams LM (2021) The role of dietary advanced glycation end products in metabolic dysfunction

For external validation, a clinically practical model incorporating clinical features and cfDNA achieved an AUC of 0.83 using linear approach

CCS, casecontrol study

We reasoned that overt oxidative stress caused by the loss of FH in non-transformed cells could instigate a tumour suppressive mechanism that could explain why Fh1 -deficient animals do not develop carcinomas but only benign cysts 19

For example, a study found that increased glutathione levels correlated with improved endothelium-dependent vasodilation, a critical factor in maintaining healthy blood flow